The bacteria cause cancer?

Microbes are everywhere around us, on our skin, in our noses and in our intestines and even in our blood and tissues.

Usually they are harmless in equilibrium with the immune system. Some are even beneficial help: bacteria in the intestines of humans in the digestion of food and produce vitamins move toxic pathogens. In fact, the human body contains more bacterial cells than somatic (body) cells.

The mitochondria, the organelles that produce energy inside human cells, have their own DNA and are probably the descendants of free-living bacteria. The bacteria are highly integrated with the functions of the whole human body.

The medical community is aware now ready to accept that perhaps certain types of bacteria or viruses, in fact, responsible for certain forms of cancer such as Kaposi’s sarcoma, stomach cancer and cervical but they are not willing to recognize that infectious agents may be inextricably linked to the development of most other tumors as well.

However, there is evidence of over a hundred years, a bacterial cause of cancer, a pleomorphic (many studies), bacteria associated with mycoplasma or similar to that observed in the slides of tumors in many respects.

In the early 20th century, the bacterial origin of cancer has been considered a dominant theory, and articles have been published about it in The Lancet. However, it was eventually shelved, despite a large number of relevant documents.

During the past century, hundreds of independent researchers of a link between bacteria and cancer in animals and humans have found, but their results have been treated as a scientific curiosity, and rarely followed by the establishment of general medicine .

But the theory has never disappeared, and scientists continue to seek ways to identify suspects and eliminate bacteria.

In 1890, German physician and bacteriologist Robert Koch, a criterion standard still in use today made to assess whether a specific bacterium is the cause of a particular disease.

“Koch’s postulates,” which is not always valid, is a useful benchmark for investigators of the disease.

Koch’s postulates are as follows:

The bacteria must be present in all cases of the disease.

The bacteria must be isolated from the host to the disease and has grown in pure culture.

The specific disease can not be reproduced when a pure culture of bacteria in a healthy host susceptible vaccinated.

The bacteria must be recoverable from the experimentally infected host.

However, Koch’s postulates have limitations, which recognized leader. They may not hold if:

The specific bacteria (such as that which causes leprosy) can not “grown in pure culture” in the laboratory.

subjects tested on animals are safe from infection.

In addition, a usually harmless bacteria cause disease if:

It acquired additional virulence factors make pathogen.

He reaches deep tissues due to trauma, surgery, an IV, etc.

It infects a patient with a vulnerable immune system.

Not everyone infected by bacteria to develop a serious disease, subclinical infection of low quality may be more common than clinically apparent, symptomatic infection.
The different types of pathogens associated with various cancers fit well enough Koch’s postulates, because they can be isolated from tumors and produce grown in petri dishes or cell culture, and sometimes, when injected into tumors in laboratory animals.

However, many are also found in lower concentrations in healthy subjects, and it appears that these microbes do not cause disease if the host is weakened.

The host’s immune system can limit the amount of infection to cause damage. For example, infection with H. pylori may lead to stomach ulcers and stomach cancer of the stomach, but many people are asymptomatic door. Not all women who developed HPV-infected cervical cancer.

Similarly, do not expect that all holders of other microbes “cancer” of getting sick. Moreover, these bacteria have the ability to produce other diseases as cancer, because H. pylori can cause stomach ulcers as well.

History

Probably the first official mention of the microbe of cancer, on 3 December 1890 Produced in William Russell, a pathologist at the School of Medicine, Royal Infirmary of Edinburgh, an address is the pathological Society of London. He described the histopathological findings, “a characteristic organism of cancer ‘that was observed under the microscope of tissue sections stained with fuchsin-all forms of cancer, which he considered, and some cases of tuberculosis, syphilis and skin infections.

The germ is both within cells and tissues, and given the size varies from barely visible to one and a half times the size of a red blood cell. Russell said that the size of some of these organisms suggestive of a fungal or yeast infection was.

Russell provisionally the microbe a possible “Blastomycetes” (a type of fungus), and called round shapes “fuchsin body” because of its blue-red staining qualities.
Nine years later, in 1899, Russell published a report published in The Lancet, the parasite of cancer “and said that to find the suspect bacteria in diseases other than the presence of cancer presents a stumbling block” the idea of a definitive function for the organism.

Cultures revealed many types of bacteria and bacterial injection in animals has given ambiguous results. Subsequently, many scientists concluded that Russell bodies were merely the result of cellular degeneration.

killing in the 1920s and 1930s, pioneering scientists Royal Raymond Rife, the use of high frequency devices and bacteria. Rife discovered that a certain range of radio waves, was lethal to bacteria, any danger to human tissue. He also invented a new form of microscope, the monochromatic light used, and was just enough to see the virus without the use of electron microscopy.

Working with a laboratory in La Jolla in the 1930s, Rife claimed a rate of 100 percent success in the treatment of cancer. Rife’s lab was due to political pressure by the American Medical Association, turning most of his works were destroyed, and currently the only known example of its microscopes in a museum.

to kill the discovery of Rife devices, high frequency of bacteria was by Hulda Clark, a Canadian researcher, work began in the 1960s picked up. Clark also said that many other diseases, like diabetes, allergies, epilepsy, Crohn’s disease, bipolar disorder, schizophrenia, which are caused by bacteria and parasites such as flukes caused.

It improved on the Rife technology, and invented a small device raidofrequency she called the Zapper it claims eliminate bacteria and other parasites from the body. Instructions for the construction of the devices have been available to the public and can be found on the Internet today.

Clark has been harassed by U.S. authorities until the establishment of his cancer clinic in Mexico, where in 2001 the authorities forbade him to offer an alternative cancer treatment left. Like Rife, Clark asked a very high success rate for treating cancer, nearly 100 percent, but no independent analysis of their claims or Rife exist.

In 1960, Dr. Virginia Livingston upset the scientific establishment by pretending to find the germs responsible for causing cancer, called “progenitor cryptocides, called the” hidden killer “. It found that the microbe is an intrinsic function was symbiotic in the human body that is responsible for initiating and life for the healing of tissues, and that the microbe was ultimately responsible for any degeneration and death of all life.

If the body has been injected into farm animals, it caused tumors to develop in some but not all subjects.
In 1974, Livingstone was first scientist to discover that the bacteria produce cancer cells and cancer cells of the hormone HCG human. This hormone is normally secreted by a human fetus in order to protect against the maternal immune system, protects against cancer of the immune attack.

Livingstone concluded that bacteria secrete factors such as mutagens actinomycin-D with cell damage human DNA, and they also exchange genetic material, such as factors of bacterial growth in human cells. Vaccines targeting produces HCG and cancer promoting bacteria, cancer cells escape a major source of HCG .. As the level of HCG will be reduced to start the immune system increases the ability to attack cancer cells.

Livingstone livestock patients themselves bacteria in blood and urine tests to stimulate the creation of “Auto Generate” vaccines, the immune system. She has published numerous articles and books, such as cancer, a new breakthrough “(1972),” The Microbiology of Cancer (1977) and “The Conquest of Cancer” (1984).

Their research was supported by other scientists as microbiologist Eleanor Alexander-Jackson, cell cytologist Irene Diller, biochemist Florence Seibert and Alan Cantwell dermatologist, among others, have been confirmed.

Milton Wainwright, a microbiologist at the University of Sheffield, United Kingdom, has written extensively on the bacteriology of cancer in recent publications as “nanobacteria and associated ‘elementary bodies in human disease and cancer” (1999) “The Return of the germ of cancer; Forgot Microbiology – Back to the Future” (2000), pleomorphic “highly staphylococci as a cause of cancer” (2000) and “Is the cancer germ ‘historic’? ( 2003).

Currently, one of the most famous proponents of the link between cancer and popular bacteria, Dr. Alan Cantwell, numerous articles and books, has written on the subject. Cantwell isolated and reported the bacterial cell wall-deficient breast cancer, Kaposi’s sarcoma and Hodgkin’s disease. He said: “If a disease like cancer is indeed caused by microscopic bacteria that show, the doctors could not see what is clearly observed for some scientists the nineteenth and twentieth century single optical microscopy.

. And with a powerful electron microscope, it is now little excuse for not “see” bacteria ”

Mycoplasma

Mycoplasma, the oldest of the suspected bacterial theory of cancer has made as a direct cause or a signficant cofactoer in a number of other inflammatory and degenerative diseases associated.

Mycoplasmas are common in the oral and urinary tract found in healthy subjects, the women four times more likely than men to be infected by accident incidence rate same gender bias as rheumatoid arthritis, fibromyalgia, fatigue and other chronic autoimmune diseases.

In 1997, the National Centre for Emerging Infectious Diseases, Centers for Disease Control and Prevention journal, infectious diseases, articles mycoplasmas: Sophisticated, reemerging, and burdened by its reputation, by Dr. Baseman and Tully who stated :

“However, mycoplasmas are acute and chronic, even on multiple sites with complications due to extensive and brought in as co-factors associated with the disease.

Recently, mycoplasmas have cofactor in the pathogenesis of AIDS and malignant transformation, chromosomal aberrations were associated syndrome, Gulf War and other unexplained illnesses and complex, including chronic fatigue syndrome, illness Crohn’s disease and various arthritides. ”

The first strains of Mycoplasma were from animals with arthritis and lung disease isolated in 1898 at the Pasteur Institute. The first human strain was isolated in 1932 from an abscess wall.

The first connection between the mycoplasmas have been identified as the cause of rheumatic diseases in 1939 by Dr. Swift and Brown. In the late 1950s, a specific strain was identified as a cause of atypical pneumonia, and named Mycoplasma pneumoniae.

The association between immunodeficiency and autoimmune disorders with mycoplasmas was first located in the mid 1970s in patients with primary hypogammaglobulinemia (an autoimmune disease) caused by infection with four species of mycoplasmas in joint tissues.

Since then, more than 100 different species Mycoplasma been identified and reported in plants, animals and humans.
There are hundreds of studies by scientists around the world that combines different types of mycoplasma by cancer.

The research by Dr. Shy-Chung Lo Institute of Pathology Armed Forces in Washington, DC, confirmed the multi-stage malignant transformation of embryonic cell lines exposed to persistent Mycoplasma infection and animal models are so exposed.

Studies published by PJ Chan, Gynecologic Oncology (1996), “The oncogenic potential of mycoplasmas has been achieved recently when they revealed chromosomal changes and cause progressive cell transformation in vitro through progressive chromosomal translocations loss. “Chan and colleagues also report on the prevalence of mycoplasma DNA in ovarian cancer 1

In 1993, a research team analyzed by C. Ilantzis at McGill Cancer Centre, Montreal, Canada conducted as part of cancer markers that are specific to different organs in the body. These markers, called “organ-specific neoantigens (NSOs), trigger the specific immune response. After the analysis of proteins OSN human colon adenocarcinomas, the researchers found the NSOs in origin2 mycoplasma

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(1). DNA Chan PJ et al Prevalence of mycoplasma conserved Detected in malignant ovarian cancer with sensitive PCR-ELISA gynecologist oncologist 1996, pp. 258-260 (3)

(2). C Ilantzis, DM Thomson, Michaelidou, S Benchimol Identification of a human organ-specific cancer neoantigen Microbiol Immunol 1993; 37 (2) :119-28

Microscopy

The “cancer bacteria” have a varied appearance, both in tissues and in culture. They may appear as cocci (spheres) 0.1 microns (one micron is 1 / 1000 mm), called “submicroscopic” because they are still considered by an ordinary microscope.

Scientists have used the term “nanobacteria” is used to describe very small bacteria from 0.05 to 0.2 microns wide size. Viruses, which measure 0.01 to 0.02 micrometers, can be viewed with an electron microscope. The smallest forms of bacteria easily through a standard filter of high porosity with viral 0 .2 microns, microbiologists assumed (until recently) is that all bacteria, firms tend to be much larger.
Once these are tiny cocci in a petri dish and the resulting culture observed over time, bacteria also produce large sticks and branches, bits of mushrooms.

Mycobacteria are known in various forms, and the microbe, Mycobacterium tuberculosis, is a good example of this complex life cycle. Some forms of the bacillus are round “coccoid” forms, other forms are typically “acid-resistant” and “rod” forms. All mycobacteria form a phylogenetic link or bridge between the bacteria and the “more” of mushrooms. “Myco” is Greek for fungi. This is the origin of the concept of mycobacteria. Mycoplasma as a plasma that is flowing structure of the cell wall – hence “plasma”.

Contrary to common bacteria, is the suspicion of cancer microbe Mycoplasma lack a cell wall. It penetrates tissue cells, and uses the cell to reproduce, as a retrovirus. If Mycoplasma bursts the cell, it takes a piece of the host cell membrane with it. When the immune system attacks the Mycoplasma, it may also be wrong to attack the host cell, leading to an autoimmune disease. question can not enter cells, natural killer immune system, immune system disorders. Because he can hide at the back of cells, it is extremely difficult to detect and eliminate.

Treatment with antibiotics Mycoplasma

Antibiotic treatment should be tailored to specific bacterial infection. Many bacteria, particularly mycoplasmas are resistant to many common antibiotics. However, a number of targeted therapies that have been known to kill specific cancer-causing bacteria to be effective, at least in the early stages of the disease.

mycoplasmal infections are treatable with long cycles of high dose antibiotics such as doxycycline and tetracycline, followed by a long period of low dose antibiotics. Because of their lack of mycoplasma from the cell wall are not affected by penicillin. Because the organism is slow growing, intracellular type with a long life cycle, several long-term course of antibiotics may be needed. The infection can be there for several months or years are treated essentially the same protocol as for Lyme disease.
No clinical studies have been published regarding the treatment of cancer with antibiotics against mycoplasma.

Vaccines against Mycoplasma

Maruyama vaccine is similar to the BCG vaccination, which are both made of mycobacteria tuberculosis isolates. Both have been widely used as a stimulant of the immune system in patients with cancer. Murayama vaccine is made of mycobacteria tuberculosis isolates, and BCG is derived from a bacillus of bovine tuberculosis attenuated. However, BCG has more side effects than the vaccine Maruyama.

Maruyama vaccine, Dr. Chisato Maruyama was invented more than 50 years, may be used alone or in combination with standard therapies. Some Japanese doctors claim that a complete remission achieved in cancers with poor prognosis, but there are no large clinical study. No negative side effects of the vaccine have been reported.

Murayama vaccine approved by the FDA for the treatment of patients with terminal cancer. Some forms of health insurance covers the costs if the vaccine is used as part of standard therapy, as it is officially approved only as an immune stimulant to counteract the side effects of bone marrow in radiotherapy.

Maruyama vaccine by the Research Institute of vaccine therapy for tumors and infections diseases, Nippon Medical School Hospital in Tokyo delivered unless the patient makes a request from their doctor. It is not expensive, about 9000 yen (100 USD) for a course of 40 days of treatment.

According to an article published in Cancer Detection and Prevention, 2003, by Tetsuo Kimoto MD, Ph.D., Maruyama vaccine did not direct cytotoxic effect on tumors, but their causes encapsulation of collagen fibers.

This leads to the reduction and partial necrosis (death) of tumors and their metastases. The survival time increased in both animals and humans to tumors, and explained that patients Kimoto Murayama vaccines, whose tumors are inoperable and resistant to conventional chemotherapy benefit. ’1

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(1). Tetsuo Kimoto MD, Ph.D. The anti-tumor Maruyama vaccine (SSM) detection of cancer and prevention Volume 22 Number 4340 Page – August 1998.

Herpes

Cancer of the cervix, caused by human papilloma virus, strikes more than 10,000 women each year in the United States, with more than 3,700. A new vaccine against the virus, Gardasil was approved by the FDA in 2006. The vaccine is effective against HPV types 16 and 18 cause about 70 percent of cervical cancers and against HPV types 6 and 11 cause about 90 percent of genital warts.

Less known is the fact that HPV is also implicated in squamous cell head and neck, especially cancer of the tonsils. 1.2 Centre researchers at Johns Hopkins University Oncology tested tumor tissue from 253 patients with tumors of head and neck and found 25 per cent of cases were HPV-positive. In 90 percent of HPV-positive tumors, HPV16, the type of virus most commonly associated with cancer of the cervix present.3

Several studies confirm the link between HPV and head and neck tumors. Approximately 31,000 people in the United States are diagnosed annually with cancer of the oral cavity and pharynx, causing 8,500 deaths per year.

The vaccine against HPV only works when given before infection, emphasizing the importance of immunization before potential exposure to the virus. Also, Gardasil does not protect against less common HPV types to protect, not included in the vaccine, which the routine and regular pap screening remain critically important to detect precancerous changes in the cervix of therapy may be used to point against cancer of the cervix uteri. This is a preventive measure, not a treatment for existing cervical or head and neck tumors.

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(1). IB Paz et al. Papillomavirus (HPV) in head and neck tumors. An association of HPV 16 with squamous cell carcinoma of Waldeyer’s tonsillar ring. First cancer in February 1997, 79 (3) :595-604th

(2) Klussman JP et al. Carcinomas of human papillomavirus positive tonsil: a different tumor entity? Med Microbiol Immunol (Berlin) 2003 Aug; 192 (3) :129-32. Epub 2002 September 14.

(3). Gillison ML, Koch WM, Capone RB, Spafford M, Westra WH, Wu L, Zahurak ML, Daniel RW, Viglione M, Symer DE, Shah KV, Sidransky D, proof of a causal relation between human papillomavirus and a subset of head and neck cancer. Journal of the National Cancer Institute. 2000 May 3, 1992 (9) :709-20

Stomach Cancer

In December 2000 edition of the Journal of the National Cancer Institute, a research team led by Pelayo Correa led Colombian pathologists indicated that antibiotics, vitamin C or beta-carotene (provitamin A) can reverse gastric precancerous conditions caused by Helicobacter pylori.

Stomach cancer is the second most common cancer worldwide the most common and is more common in countries like Colombia and China, where H. pylori infects more than half the population in early childhood. In the United States, where H. pylori is less common, rates of stomach cancer have declined since the 1930s.

The two main risk factors for stomach cancer is infection with H. pylori and a diet that is low in vitamin C and beta-carotene, which the body converts into vitamin A. He also ample evidence that a diet with fresh fruits and vegetables that are rich in these nutrients, protects against cancer of the stomach.
In 1992, researchers studied 631 patients with gastric aberrant cell growth, cancerous growths in one of three consecutive phases – nonmetaplastic multifocal atrophy, intestinal metaplasia falls, and dysplasia.

Patients received either a placebo pill, a supplement of vitamin C or beta-carotene, or antibiotics against H. pylori. Others received a combination of medicines and food supplements.
Scientists have gastric biopsies of patients after 3 and 6 years of treatment. Patients with atrophy were about five times more likely to decline in the growth of precancerous cells present as always, experience placebo.

Among those with metaplasia, were volunteers, ingestion of food supplements or drugs were three times more likely than placebo were better. However, patients with dysplasia, the last stage of gastro-intestinal cancer, showed no significant improvement with one treatment. “The earlier in the process of [us] more chance of regression to intervene,” said Correa. 1

This study is encouraging because it shows that the treatment of cancer-causing bacteria produced clear benefits against precancerous conditions. However, once tissue damage caused by an infection that has progressed precancerous stage, antibiotics produced no benefit and would probably not improve in cancer cases, be purely and simply.

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(1). Correa, P., et al. 2000th Chemoprevention of gastric dysplasia: randomized trial of antioxidant supplements and anti-Helicobacter pylori. Journal of the National Cancer Institute 92 (December 6) :1881-1888

Lymphoma

The common antibiotic doxycycline effectively treats a type of lymphoma in ocular chlamydial infection, according to a study published in the October 4 issue of the Journal of the National Cancer Institute published.

A team of researchers from Andres JM Ferreri, MD, San Raffaele Scientific Institute H resulted in Milan, Italy, gave 27 patients with ocular adnexal lymphoma (UT) is a 3-week course of doxycycline therapy, whether positive or negative for chlamydia tested.

Researchers have observed for tumor progression every 6 months, and found that doxycycline causes causes of lymphoma patients regardless of recourse if they tested positive or negative.
The study suggests that doxycycline has a useful therapy in patients without other treatment, and it is a valid alternative to chemotherapy and radiotherapy, without the same toxic side effects. Patients who were treated with doxycycline, a rate of 66% disease-free survival. 1

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(1). Andrés JM Ferreri, Maurilio Ponzoni, Massimo Guidoboni, Antonio Giordano Resti, Letterio S. Politi, Sergio Cortelazzo, Judit Demeter, Francesco Zallio, Angelo Palmas, Giuliana Muti, Giuseppina P. Dognin, Elisa Pasini, Antonia Anna Lettini, Federico Sacchetti, Carlo De Conciliis Claudio DOGLIONI, Riccardo Dolcetti Bacteria-eradicating therapy with doxycycline in ocular adnexal MALT lymphoma: A prospective multicenter study, Journal of the National Institute of Cancer 2006 98 (19) :1375-1382

Note on the indiscriminate use of antibiotics

So far, no treatment has been discovered with antibiotics that are effective in treating most types of cancer, and a study linking the use of antibiotics with an increased risk of breast cancer published in the Journal February 2004 of the American Medical Association. The study, 10,000 women in Washington State found that those who took more than 25 courses of two antibiotics over an average of 17 years had the risk of breast cancer compared to women who tested without antibiotics. Women who have between one and 25 prescriptions over the same period was one and a half times increased risk of breast cancer. 1

Correlation does not always mean causation, and this study raises interesting questions about the mechanism of this effect. Perhaps this is due to cell damage directly caused by the antibiotic.